Hacker News ( unofficial )
HN Discussion: https://news.ycombinator.com/item?id=17128413
Posted by ColinWright (karma: 88891)
Post stats: Points: 153 - Comments: 53 - 2018-05-22T18:47:54Z
#HackerNews #end #flares #iridium #the
HackerNewsBot debug: Calculated post rank: 119 - Loop: 411 - Rank min: 100 - Author rank: 71
Hacker News ( unofficial )
It's with sad hearts that we are announcing that search.cpan.org will be retired on the 25th of June 2018. Graham Barr originally wrote t...
Article word count: 307
HN Discussion: https://news.ycombinator.com/item?id=17128162
Posted by jasonjayr (karma: 625)
Post stats: Points: 188 - Comments: 43 - 2018-05-22T18:18:48Z
#HackerNews #end #era #goodbye #saying #searchcpanorg #the
Article content:
Itʼs with sad hearts that we are announcing that [1]search.cpan.org will be retired on the 25th of June 2018. Graham Barr originally wrote the site nearly 20 years ago -- it first went live in early 1999 -- and it quickly became an invaluable resource for Perl developers around the world.
The ability to search [2]CPAN (Comprehensive Perl Archive Network) and read Perl module documentation online helped spark many developers interest in Perl and helped to build the Perl community.
The site was originally hosted by [3]Washington University in St. Louis on a single Solaris box. For his work on search.cpan.org, Graham won a [4]White Camel award in 2002.
The site was later moved to the [5]Perl NOC. US mirrors were hosted by [6]YellowBot and [7]Phyber and a European mirror has been hosted by [8]digital craftsmen for the last 10 years. These amazing people and companies helped make the site a success.
In recent years maintenance has become a burden. Most of the site is running 2005 era Perl code. Luckily, there is now a viable alternative: [9]MetaCPAN.org. The MetaCPAN team has been getting ready for the transition and is nearly ready to take over.
Shortly, a link will be added to all pages on search.cpan.org to inform users of the upcoming change. After about a month, all traffic will be redirected to the equivalent MetaCPAN page. We would like to thank all who have assisted with this project. An extra special thanks to Graham for his hard work and unwavering support of search.cpan.org for all these years. To search.cpan.org -- we will miss you.
Update #1 (2018-05-19): Thereʼs some confusion around whether or not existing search.cpan.org URLs will continue to work. They will! The vast majority of of them will be transparently served by MetaCPAN pages. You do not need to update any links right now.
References
Visible links
1. http://search.cpan.org/
2. http://www.cpan.org/
3. http://www.wustl.edu/
4. https://www.perl.org/advocacy/white_camel/2002.html
5. https://log.perl.org/
6. http://www.yellowbot.com/
7. https://www.phyber.com/
8. http://digitalcraftsmen.com/
9. https://metacpan.org/
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Hacker News ( unofficial )
The genome obviously varies from person to person. But it can also vary from cell to cell, even within the same individual. The implications of “mosaicism” are enormous.
Article word count: 2132
HN Discussion: https://news.ycombinator.com/item?id=17126796
Posted by bookofjoe (karma: 850)
Post stats: Points: 106 - Comments: 109 - 2018-05-22T15:46:17Z
#HackerNews #cell #from #genome #mosaicism #the #varies
Article content:
[1]Matter
CreditJason Holley
James Priest couldn’t make sense of it. He was examining the DNA of a desperately ill baby, searching for a genetic mutation that threatened to stop her heart. But the results looked as if they had come from two different infants.
“I was just flabbergasted,” said Dr. Priest, a pediatric cardiologist at Stanford University.
The baby, it turned out, carried a mixture of genetically distinct cells, a condition known as mosaicism. Some of her cells carried the deadly mutation, but others did not. They could have belonged to a healthy child.
We’re accustomed to thinking of our cells sharing an identical set of genes, faithfully copied ever since we were mere fertilized eggs. When we talk about our genome — all the DNA in our cells — we speak in the singular.
But over the course of decades, it has become clear that the genome doesn’t just vary from person to person. It also varies from cell to cell. The condition is not uncommon: We are all mosaics.
For some people, that can mean developing a serious disorder like a heart condition. But mosaicism also means that even healthy people are more different from one another than scientists had imagined.
Magical Mystery
In medieval Europe, travelers making their way through forests sometimes encountered a terrifying tree.
A growth sprouting from the trunk [2]looked as if it belonged to a different plant altogether. It formed a dense bundle of twigs, the sort that people might fashion into a broom.
Germans call it Hexenbesen: witches’ broom. As legend had it, witches used magic spells to conjure the brooms to fly across the night sky. The witches used some as nests, too, leaving them for hobgoblins to sleep in.
In the 19th century, plant breeders found that if they cut witches’ broom from one tree and grafted it to another, the broom would grow and produce seeds. Those seeds would sprout into witches’ broom as well.
Today you can see examples of witches’ broom on ordinary suburban lawns. Dwarf Alberta spruce is a landscaping favorite, growing up to ten feet high. It comes from northern Canada, where botanists in 1903 discovered the first known dwarf clinging to a white spruce — a species that can grow ten stories tall.
Pink grapefruits [3]arose in much the same way. A Florida farmer noticed an odd branch on a Walters grapefruit tree. These normally bear white fruit, but this branch was weighed down with grapefruits that had pink flesh. Those seeds have produced pink grapefruit trees ever since.
Charles Darwin was fascinated by such oddities. He marveled at reports of “bud sports,” strange, atypical blooms on flowering plants. Darwin thought they held clues to the mysteries of heredity.
The cells of plants and animals, he reasoned, must contain “particles” that determined their color, shape and other traits. When they divided, the new cells must inherit those particles.
Something must scramble that heritable material when bud sports arose, Darwin declared, like “the spark which ignites a mass of combustible matter.”
Only in the 20th century did it become clear that this combustible matter was DNA. After one cell mutates, scientists found, all its descendants inherit that mutation.
Witches’ broom and bud sports eventually came to be known as mosaics, after the artworks made up of tiny tiles. Nature creates its mosaics from cells instead of tiles, in a rainbow of different genetic profiles.
Before DNA sequencing was commonplace, scientists struggled to tell the genetic differences between human cells. Cancer offered the first clear evidence that humans, like plants, could become mosaics.
In the late 1800s, biologists studying cancer cells noticed that many of them had oddly shaped chromosomes. A German researcher, Theodor Boveri, speculated at the turn of the century that gaining abnormal chromosomes [4]could actually make a cell cancerous.
As soon as Boveri floated his theory, he faced intense opposition. “The skepticism with which my ideas were met when I discussed them with investigators who act as judges in this area induced me to abandon the project,” he later said.
Boveri died in 1914, and it took nearly five decades for scientists to discover he was right.
David A. Hungerford and Peter Nowell found that people with a form of cancer called chronic myelogenous leukemia were missing a substantial chunk of chromosome 22. It turned out a mutation had moved that chunk over to chromosome 9. The cells that inherited that mutation became cancerous.
It’s hard to think that a tumor might have anything in common with a pink grapefruit. Yet they are both products of the same process: lineages of cells that gain new mutations not found in the rest of the body.
Some skin diseases [5]proved to be caused by mosaicism, too. Certain genetic mutations cause one side of the body to become entirely dark. Other mutations draw streaks across the skin.
The difference is in the timing. If a cell gains a mutation very early in development, it will produce many daughter cells that will end up spreading across much of the body. Late-arising mutations will have a more limited legacy.
Dr. Walsh and his colleagues have found evidence of mosaicism in some very unexpected places.
They investigated a mysterious disorder called hemimegalencephaly, which causes one side of the brain to become overgrown. The researchers [6]examined tissue from patients who had brain surgery to treat the seizures triggered by hemimegalencephaly.
Some of the brain cells in the patients — but not all of the cells — shared the same mutant genes. It’s possible that these mutant neurons multiplied faster than others in the brain, triggering one side to become enlarged.
Preliminary studies suggest that mosaicism [7]underlies many other diseases. Last year, Christopher Walsh, a geneticist at Harvard University, and his colleagues published evidence that mosaic mutations [8]may raise the risk of autism.
But scientists are also finding that mosaicism does not automatically equal disease. In fact, it’s the norm.
When a fertilized egg — known as a zygote — starts dividing in the womb, many of its early descendant cells end up with the wrong number of chromosomes. Some are accidentally duplicated, and others lost.
Most of these unbalanced cells divide only slowly or die off altogether, while the normal cells multiply far faster. But a surprising number of embryos survive with some variety in their chromosomes.
Markus Grompe, a biologist at Oregon Health & Science University, and his colleagues looked at liver cells from children and adults without liver disease. Between a quarter and a half of the cells [9]were aneuploids, typically missing one copy of one chromosome.
Image
CreditJason Holley
Along with altered chromosomes, human embryos also gain smaller mutations in the genome. Stretches of DNA may be copied or deleted. Single genetic letters may get incorrectly reproduced.
It wasn’t possible to study such molecular changes accurately until DNA-sequencing technology became sophisticated enough.
In 2017, researchers at the Wellcome Trust Sanger Institute in England examined 241 women, sequencing batches of white blood cells from each. Every woman [10]had acquired about 160 new mutations, each present in a sizable fraction of her cells.
The women gained these mutations as embryos, the scientists theorized, with two or three new mutations arising each time a cell divided. As those new mutations occurred, the embryonic cells passed them all down to their descendants, a mosaic legacy.
Dr. Walsh and his colleagues have discovered intricate mosaics in the brains of healthy people. In one study, they plucked neurons from the brain of a 17-year-old boy who had died in a car accident. They sequenced the DNA in each neuron and compared it to the DNA in cells from the boy’s liver, heart and lungs.
Every neuron, the researchers found, had hundreds of mutations not found in the other organs. But many of the mutations were shared only by some of the other neurons.
It occurred to Dr. Walsh that he could use the mutations to reconstruct the cell lineages — to learn how they had originated. The researchers used the patterns to draw a sort of genealogy, linking each neuron first to its close cousins and then its more distant relatives.
When they had finished, the scientists found that the cells belonged to five main lineages. The cells in each lineage all inherited the same distinctive mosaic signature.
Even stranger, the scientists found cells in the boy’s heart with the same signature of mutations found in some brain neurons. Other lineages included cells from other organs.
Based on these results, the researchers [11]pieced together a biography of the boy’s brain.
When he was just an embryonic ball in the womb, five lineages of cells had emerged, each with a distinct set of mutations. Cells from those lineages migrated in different directions, eventually helping to produce different organs — including the brain.
The cells that became the brain turned into neurons, but they did not all belong to the same family. Different lineages merged together. In essence, the boy’s brain was made of millions of mosaic clusters, each composed of tiny cellular cousins.
It’s hard to say what these mosaic neurons mean to our lives — what it means for each of us to have witches’ broom growing in our skulls. “We don’t know yet whether they have any effect on shaping our abilities or challenges,” said Dr. Walsh.
What we do know is that mosaicism introduces randomness into the development of our brains. Mutations, which arise at random, will form different patterns in different people. “The same zygote would never develop exactly the same way twice,” said Dr. Walsh.
As ubiquitous as mosaicism may be, it’s still easy to overlook — and surprisingly hard to document.
Astrea Li, the infant examined by Dr. Priest at Stanford, had gone into cardiac arrest the day she was born. Her doctors put a defibrillator in her heart to shock it back into the proper rhythm.
Dr. Priest sequenced Astrea’s genome to search for the cause of her disorder. He concluded that she had a mutation in one copy of a gene called SCN5A. That mutation could have caused her trouble, because it encodes a protein that helps trigger heartbeats.
But when Dr. Priest ran a different test, he couldn’t find the mutation.
To get to the bottom of this mystery, he teamed up with Steven Quake, a Stanford biologist who had pioneered methods for sequencing the genomes of individual cells. Dr. Priest plucked 36 white blood cells from the child’s blood, and the scientists sequenced the entire genome of each cell.
In 33 of the cells, both copies of a gene called SCN5A were normal. But in the other three cells, the researchers found a mutation on one copy of the gene. Astrea had mosaic blood.
Her saliva and urine also turned out to contain mosaic cells, some of which carried the mutation. These findings demonstrated that Astrea had become a mosaic very early in her development.
The skin cells in her saliva, the bladder cells in her urine and her blood cells each originated from a different layer of cells in two-week-old embryos.
Astrea’s SCN5A mutation must have originated in a cell that existed before that stage. Its daughter cells later ended up in those three layers, and ultimately in tissues scattered throughout her body.
They might very well have ended up in her heart, too. And there the mutation could have theoretically caused Astrea’s problems.
While Dr. Priest was reconstructing Astrea’s mosaic origins, she was recovering from the surgery to implant her defibrillator. Her parents, Edison Li and Sici Tsoi, brought her home. And for a few months, it seemed she was out of the woods.
One day, however, her defibrillator sensed an irregular heartbeat and released a shock — along with a wireless message to Astrea’s doctors.
Back at the hospital, doctors discovered a new problem: her heart had become dangerously enlarged. Researchers have linked mutations in the SCN5A gene to the condition.
Her heart soon stopped. Her doctors attached a mechanical pump, and soon a donated heart became available.
Astrea underwent transplantation surgery and recovered well enough to go home. She went on to enjoy a normal childhood, performing cartwheels with her sister and listening obsessively to the soundtrack of “Frozen.”
The transplant did not just give Astrea a new lease on life. It also gave Dr. Priest a very rare chance to look at a mosaic heart up close.
The transplant surgeons had clipped some pieces of Astrea’s cardiac muscle. Dr. Priest and his colleagues extracted the SCN5A gene from the cells taken from different parts of her heart.
On the right side of the heart, he and his colleagues [12]found that more than 5 percent of the cells had mutant genes. On the left, nearly 12 percent did.
To study the effect of this mosaicism, Dr. Priest and his colleagues built a computer simulation of Astrea’s heart. They programmed it with grains of mutant cells and let it beat.
The simulated heart thumped irregularly, in much the same way Astrea’s had.
The experience left Dr. Priest wondering how many more people might be at risk from a hidden mix of mutations.
Unless he winds up with another patient like Astrea, we may never find out.
References
Visible links
1. https://www.nytimes.com/column/matter
2. http://www.jstor.org/stable/42953803
3. https://www.researchgate.net/profile/John_Da_Graca2/publication/263596266_The_Origins_of_Red_Pigmented_Grapefruits_and_the_Development_of_New_Varieties/links/0a85e53b56a49f0cd4000000.pdf
4. http://dx.doi.org/10.1002/path.4408
5. http://dx.doi.org/10.1590/abd1806-4841.20132015
6. http://dx.doi.org/10.1126/science.1237758
7. https://www.ncbi.nlm.nih.gov/pmc/articles/pmid/28450582/
8. https://www.nature.com/articles/nn.4598
9. http://dx.doi.org/10.1053/j.gastro.2011.10.029
10. https://www.nature.com/articles/nature21703
11. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4664477
12. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5068256/
HackerNewsBot debug: Calculated post rank: 107 - Loop: 190 - Rank min: 100 - Author rank: 18
Hacker News ( unofficial )
Comcast has just been caught in a major security snafu: revealing the passwords of its customers’ Xfinity-provided wireless routers in plaintext on the web. Anyone with a subscriber’s account number…
Article word count: 445
HN Discussion: https://news.ycombinator.com/item?id=17122917
Posted by smaili (karma: 4905)
Post stats: Points: 106 - Comments: 31 - 2018-05-22T00:19:11Z
#HackerNews #and #comcast #customers #leaking #names #passwords #routers #the #wireless
Article content:
[1]Comcast has just been caught in a major security snafu: revealing the passwords of its customers’ Xfinity-provided wireless routers in plaintext on the web. Anyone with a subscriber’s account number and street address number will be served up the Wi-Fi name and password via the company’s Xfinity internet activation service.
Security researchers Karan Saini and Ryan Stevenson [2]reported the issue to ZDnet.
The site is meant to help people setting up their internet for the first time: ideally, you put in your data, and Comcast sends back the router credentials while activating the service.
The problem is threefold:
1. You can “activate” an account that’s already active
2. The data required to do so is minimal and it is not verified via text or email
3. The wireless name and password are sent on the web in plaintext
This means that anyone with your account number and street address number (e.g. the 1425 in “1425 Alder Ave,” no street name, city, or apartment number needed), both of which can be found on your paper bill or in an email, will instantly be given your router’s SSID and password, allowing them to log in and use it however they like or monitor its traffic. They could also rename the router’s network or change its password, locking out subscribers.
This only affects people who use a router provided by Xfinity/Comcast, which comes with its own name and password built in. Though it also returns custom SSIDs and passwords, since they’re synced with your account and can be changed via app and other methods.
What can you do? While this problem is at large, it’s no good changing your password — Comcast will just provide any malicious actor the new one. So until further notice all of Comcast’s Xfinity customers with routers provided by the company are at risk.
One thing you can do for now is treat your home network as if it is a public one — if you must use it, make sure encryption is enabled if you conduct any private business like buying things online. What will likely happen is Comcast will issue a notice and ask users to change their router passwords at large.
Another is to buy your own router — this is a good idea anyway, as it will pay for itself in a few months and you can do more stuff with it. Which to buy and how to install it, however, are beyond the scope of this article. But if you’re really worried, you could conceivably fix this security issue today by bringing your own hardware to the bargain.
I’ve contacted the company for comment and will update when I hear back.
References
Visible links
1. https://www.crunchbase.com/organization/comcast/
2. https://www.zdnet.com/article/comcast-bug-leaks-xfinity-home-addresses-wireless-passwords/
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Hacker News ( unofficial )
(Submitted on 4 May 2018) Abstract: The question of continuous-versus-discrete information representation in the brain is a fundamental yet unresolved physiological question. Historically, most…
Article word count: 264
HN Discussion: https://news.ycombinator.com/item?id=17123168
Posted by gballan (karma: 824)
Post stats: Points: 126 - Comments: 33 - 2018-05-22T01:32:20Z
#HackerNews #brain #continuous #discrete #form #information #represented #the
Article content:
(Submitted on 4 May 2018)
Abstract: The question of continuous-versus-discrete information representation in the brain is a fundamental yet unresolved physiological question. Historically, most analyses assume a continuous representation without considering the alternative possibility of a discrete representation. Our work explores the plausibility of both representations, and answers the question from a communications engineering perspective. Drawing on the well-established Shannonʼs communications theory, we posit that information in the brain is represented in a discrete form. Using a computer simulation, we show that information cannot be communicated reliably between neurons using a continuous representation, due to the presence of noise; neural information has to be in a discrete form. In addition, we designed 3 (human) behavioral experiments on probability estimation and analyzed the data using a novel discrete (quantized) model of probability. Under a discrete model of probability, two distinct probabilities (say, 0.57 and 0.58) are treated indifferently. We found that data from all participants were better fit to discrete models than continuous ones. Furthermore, we re-analyzed the data from a published (human) behavioral study on intertemporal choice using a novel discrete (quantized) model of intertemporal choice. Under such a model, two distinct time delays (say, 16 days and 17 days) are treated indifferently. We found corroborating results, showing that data from all participants were better fit to discrete models than continuous ones. In summary, all results reported here support our discrete hypothesis of information representation in the brain, which signifies a major demarcation from the current understanding of the brainʼs physiology.
From: James Tee [[1]view email]
[v1] Fri, 4 May 2018 07:24:50 GMT (4840kb)
References
Visible links
1. https://arxiv.org/show-email/f54b7290/1805.01631
HackerNewsBot debug: Calculated post rank: 95 - Loop: 288 - Rank min: 80 - Author rank: 61
Hacker News ( unofficial )
The folding wings will let the Boeing 777X dock at standard airport gates.
Article word count: 358
HN Discussion: https://news.ycombinator.com/item?id=17121457
Posted by mpweiher (karma: 24841)
Post stats: Points: 79 - Comments: 85 - 2018-05-21T20:14:40Z
#HackerNews #boeings #faa #folding #get #green #light #the #wingtips
Article content:
Once the 777X lands, the wingtips will rotate until they point upwards. [1]Bloomberg notes that the plane will be the only commercial model in widespread use to have such a feature. Some smaller planes have foldable wings, including [2]military aircraft that have to take up as little space as possible on aircraft carriers. NASA, meanwhile, is [3]testing folding wings that adjust midair for supersonic flights.
The 777Xʼs wingtips are so novel that US regulators had to [4]draw up new standards for them. The FAA rubber-stamped those measures Friday.
The agency was concerned that the wingtips could cause safety issues -- some plane crashes occurred after pilots did not secure flaps on wings before takeoff. The FAA required Boeing to have several warning systems to make sure pilots wonʼt attempt a takeoff before the wingtips are locked in the correct position. The FAA also wanted assurances that there was no way the tips would rotate during flight, and that the wings could handle winds of up to 75 miles per hour while on the ground.
[5]IFrame
The new wings are made from carbon-fiber composites that are stronger and lighter than the metal Boeing uses in other wings. That lets the company increase the wingsʼ width by 23 feet to 235 feet, which makes flying more efficient. These are the widest wings Boeing has attached to a plane, surpassing the 747-8ʼs 224 feet. However, it doesnʼt hold the record for a commercial plane: the Airbus A380 has a 262-foot-wide wing, which forced some airports to install gates specifically to accommodate it.
The FAAʼs decision moves the 777X closer to a commercial reality, four and a half years after the plane was announced. There was a [6]setback recently, though: there was a three-month delay in [7]starting trials of the engine, which General Electric is supplying, largely because of a compressor problem. Thereʼs been a delay with the wings too over a longer-than-expected process in crafting structural ribs, though Boeing hopes to be back on track by summer. Despite the hitches, Boeing is still working towards the 777Xʼs first flight, which is scheduled for Q1 next year. Commercial departures wonʼt take place until at least that December.
References
Visible links
1. https://www.bloomberg.com/news/articles/2018-05-18/boeing-s-sci-fi-foldable-wing-design-for-new-777s-gets-go-ahead
2. https://www.youtube.com/watch?v=1er9KSK8NeU
3. https://www.engadget.com/2018/01/25/nasa-tests-foldable-plane-wings/
4. https://s3.amazonaws.com/public-inspection.federalregister.gov/2018-10576.pdf
5. https://www.youtube.com/embed/RGsz_toWM5A
6. https://finance.yahoo.com/news/boeing-moves-keep-777x-track-010709749.html
7. https://www.engadget.com/2018/03/18/ge-test-flies-giant-ge9x-jet-engine/
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Hacker News ( unofficial )
© 2005-2018 Markus Triska Selected example programs are available as showcases. For offline access, you can download this book from a public git repository: https://github.com/triska/the-power-of…
HN link: https://news.ycombinator.com/item?id=17121028
Posted by tosh (karma: 19762)
Post stats: Points: 132 - Comments: 41 - 2018-05-21T19:23:48Z
#HackerNews #power #prolog #the
HackerNewsBot debug: Calculated post rank: 101 - Loop: 150 - Rank min: 100 - Author rank: 59
Hacker News ( unofficial )
HN link: https://news.ycombinator.com/item?id=17114563
Posted by tosh (karma: 19638)
Post stats: Points: 114 - Comments: 21 - 2018-05-20T20:20:59Z
#HackerNews #2006 #complete #history #lemmings #the
HackerNewsBot debug: Calculated post rank: 83 - Loop: 160 - Rank min: 80 - Author rank: 59
Hacker News ( unofficial )
HN link: https://news.ycombinator.com/item?id=17112302
Posted by thebillkidy (karma: 146)
Post stats: Points: 143 - Comments: 25 - 2018-05-20T11:36:14Z
#HackerNews #and #chain #decision #markov #process #property #reward #the
HackerNewsBot debug: Calculated post rank: 103 - Loop: 368 - Rank min: 100 - Author rank: 23
Hacker News ( unofficial )
HN link: https://news.ycombinator.com/item?id=17114308
Posted by tosh (karma: 19613)
Post stats: Points: 83 - Comments: 91 - 2018-05-20T19:15:39Z
#HackerNews #2017 #great #haskell #hasnt #over #taken #the #why #world
HackerNewsBot debug: Calculated post rank: 85 - Loop: 133 - Rank min: 80 - Author rank: 59
Hacker News ( unofficial )
HN link: https://news.ycombinator.com/item?id=17113095
Posted by dotcoma (karma: 1834)
Post stats: Points: 131 - Comments: 41 - 2018-05-20T15:17:53Z
#HackerNews #facebook #hearing #let #part #the
HackerNewsBot debug: Calculated post rank: 101 - Loop: 77 - Rank min: 100 - Author rank: 27
Hacker News ( unofficial )
HN link: https://news.ycombinator.com/item?id=17112670
Posted by okket (karma: 22276)
Post stats: Points: 145 - Comments: 105 - 2018-05-20T13:53:28Z
#HackerNews #1password #app #for #from #mac #only #removed #store #subscription #the #update #version
HackerNewsBot debug: Calculated post rank: 131 - Loop: 78 - Rank min: 100 - Author rank: 66
Hacker News ( unofficial )
HN link: https://news.ycombinator.com/item?id=17108578
Posted by reportingsjr (karma: 583)
Post stats: Points: 138 - Comments: 27 - 2018-05-19T16:37:44Z
#HackerNews #76477 #chip #effect #inside #invaders #sound #space #the
HackerNewsBot debug: Calculated post rank: 101 - Loop: 360 - Rank min: 100 - Author rank: 35
Hacker News ( unofficial )
HN link: https://news.ycombinator.com/item?id=17109114
Posted by fanf2 (karma: 7472)
Post stats: Points: 132 - Comments: 49 - 2018-05-19T18:43:01Z
#HackerNews #cause #cult #root #the
HackerNewsBot debug: Calculated post rank: 104 - Loop: 166 - Rank min: 100 - Author rank: 56
Hacker News ( unofficial )
HN link: https://news.ycombinator.com/item?id=17110385
Posted by dumbfounder (karma: 1106)
Post stats: Points: 192 - Comments: 35 - 2018-05-19T23:51:46Z
#HackerNews #ads #ceo #facebook #get #how #interview #reddit #targeted #the #with
HackerNewsBot debug: Calculated post rank: 139 - Loop: 54 - Rank min: 100 - Author rank: 39
Hacker News ( unofficial )
HN link: https://news.ycombinator.com/item?id=17107883
Posted by nradov (karma: 7289)
Post stats: Points: 123 - Comments: 57 - 2018-05-19T13:47:32Z
#HackerNews #20th #century #chinas #claim #emerged #first #half #islands #spratly #the
HackerNewsBot debug: Calculated post rank: 101 - Loop: 248 - Rank min: 100 - Author rank: 25
Hacker News ( unofficial )
HN link: https://news.ycombinator.com/item?id=17108291
Posted by kawera (karma: 20354)
Post stats: Points: 112 - Comments: 40 - 2018-05-19T15:37:26Z
#HackerNews #disappear #from #how #internet #the
HackerNewsBot debug: Calculated post rank: 88 - Loop: 99 - Rank min: 80 - Author rank: 75
Hacker News ( unofficial )
HN link: https://news.ycombinator.com/item?id=17107317
Posted by doener (karma: 15256)
Post stats: Points: 171 - Comments: 92 - 2018-05-19T10:37:25Z
#HackerNews #become #estonia #first #free #nation #public #the #transport #worlds
HackerNewsBot debug: Calculated post rank: 144 - Loop: 91 - Rank min: 100 - Author rank: 59
Hacker News ( unofficial )
HN link: https://news.ycombinator.com/item?id=17101830
Posted by empressplay (karma: 2750)
Post stats: Points: 232 - Comments: 46 - 2018-05-18T14:42:08Z
#HackerNews #apple #disk #efforts #engineer #floppy #his #recounts #steve #system #the #wozniak
HackerNewsBot debug: Calculated post rank: 170 - Loop: 377 - Rank min: 100 - Author rank: 31
Hacker News ( unofficial )
HN link: https://news.ycombinator.com/item?id=17103157
Posted by airesearcher (karma: 87)
Post stats: Points: 180 - Comments: 43 - 2018-05-18T17:17:02Z
#HackerNews #compression #interesting #numbers #parallax #pattern #prime #the
HackerNewsBot debug: Calculated post rank: 134 - Loop: 78 - Rank min: 100 - Author rank: 217
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